5 µm Chemotaxis Assays, 24-Well Format

  • Fully quantify chemotaxis with no manual cell counting
  • Measure chemotaxis in less than 6 hours with most cell types
  • Membrane inserts are uncoated to allow use with any chemoattractant
  • Detection with fluorescence plate reader

 

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General FAQs about Chemotaxis Assays

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CytoSelect™ 24-Well Cell Migration Assay, 5 µm
Catalog Number
CBA-102
Size
12 assays
Detection
Fluorometric
Manual/Data Sheet Download
SDS Download
Price
$435.00
CytoSelect™ 24-Well Cell Migration Assay, 5 µm
Catalog Number
CBA-102-5
Size
5 x 12 assays
Detection
Fluorometric
Manual/Data Sheet Download
SDS Download
Price
$1,960.00
CytoSelect™ 24-Well Cell Migration Assay, 5 µm, Trial Size
Catalog Number
CBA-102-T
Size
4 assays
Detection
Fluorometric
Manual/Data Sheet Download
SDS Download
Price
$215.00
Product Details

Chemotaxis describes the movement of cells toward or away from a chemical stimulus in their enviroment. Cell chemotaxis plays a pivotal role in the progression of cancer and other diseases.

CytoSelect™ Cell Migration Assays are ideal for determining the chemotactic properties of cells. The 5 µm pore size is ideal for monocytes / macrophages.

CytoSelect™ Chemotaxis Assay Principle. Migratory cells move through the polycarbonate membrane toward a chemoattractant underneath the membrane inserts.

Recent Product Citations
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  7. Sa, Y. et al. (2015). TIMP-1 induces α-smooth muscle actin in fibroblasts to promote urethral scar formation. Cell Physiol Biochem. 35:2233-2243.
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  10. Lee, Y. H. et al. (2014). Stretch-induced human myometrial cytokines enhance immune cell recruitment via endothelial activation. Cell Mol Immunol. doi: 10.1038/cmi.2014.39.
  11. Cizkova, D. et al. (2014). Modulation properties of factors released by bone marrow stromal cells on activated microglia: an in vitro study. Sci Rep. 4:7514.
  12. Jelacic, T.M., et al. (2014). Exposure to Bacillus anthracis capsule results in suppression of human monocyte-derived dendritic cells. Infect Immun. 82:3405-3416.
  13. Kiss, J. et al. (2012).Loss of the Oxygen Sensor PHD3 Enhances the Innate Immune Response to Abdominal Sepsis. J. Immunol. 189:1955-1965 (#CBA-102).
  14. Rayner, K. et al. (2008). Extracellular Release of the Atheroprotective Heat Shock Protein 27 is Mediated by Estrogen and Completely Inhibits acLDL Binding to Scavenger Receptor-A. Circ. Res. 103(2):133-141 (#CBA-102).
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