96-Well Chemotaxis / Cell Invasion Assay Combo Kits

96-Well Chemotaxis / Cell Invasion Assay Combo Kits
  • Fully quantify chemotaxis and cell invasion with no manual cell counting
  • Includes two plates with 8 µm membrane inserts: one uncoated for chemotaxis and one precoated on top of the membrane with ECM matrix (basement membrane) for cell invasion
  • Quantitation with fluorescence plate reader

 

General FAQs about Chemotaxis Assays

General FAQs about Cell Invasion Assays

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CytoSelect™ 96-Well Cell Migration and Invasion Assay Combo Kit, 8 µm
Catalog Number
CBA-106-C
Size
96 migration assays + 96 invasion assays
Detection
Fluorometric
Manual/Data Sheet Download
SDS Download
Price
$995.00
Product Details

If you are assaying both invasive and migratory properties of your cells, order one of our economic CytoSelect™ Cell Migration  / Invasion Assay Combo kits. These kits save you money compared to buying separate chemotaxis and cell invasion kits.

Each 96-well combo kit provides sufficient reagents to perform 96 cell migration plus 96 cell invasion assays.

Migration of Human Fibrosarcoma HT-1080 Cells.Cells were seeded at 30,000 cells per well of a 24-well plate and allowed to migrate toward 10% FBS for 4 hours in either the presence or absence of 2µM Cytochalasin D. Migratory cells on the bottom of the polycarbonate membrane were stained (top) or quantified in a fluorescence plate reader (bottom).

Recent Product Citations
  1. López-Moncada, F. et al. (2019). Secreted protein acidic and rich in cysteine (SPARC) induces epithelial-mesenchymal transition, enhancing migration and invasion, and is associated with high Gleason score in prostate cancer. Asian J Androl. doi: 10.4103/aja.aja_23_19.
  2. Wang, W. et al. (2019). Deoxypodophyllotoxin inhibits cell viability and invasion by blocking the PI3K/Akt signaling pathway in human glioblastoma cells. Oncol Rep. 41(4):2453-2463. doi: 10.3892/or.2019.7016.
  3. Orellana-Serradell, O. et al. (2018). The transcription factor ZEB1 promotes an aggressive phenotype in prostate cancer cell lines. Asian J Androl. 20(3):294-299. doi: 10.4103/aja.aja_61_17.
  4. Zecchini, V. et al. (2014).  Nuclear ARRB1 induces pseudohypoxia and cellular metabolism reprogramming in prostate cancer. EMBO J. 33:1365-1382.
  5. Sharma, N. L. et al. (2014).  The ETS family member GABPα modulates androgen receptor signalling and mediates an aggressive phenotype in prostate cancer. Nucleic Acids Res. 42:6256-6269.
  6. Zecchini, V. et al. (2014).  Nuclear ARRB1 induces pseudohypoxia and cellular metabolism reprogramming in prostate cancer. EMBO J. 33:1365-1382.
  7. Ardiani, A. et al. (2014). Vaccine-mediated immunotherapy directed against a transcription factor driving the metastatic process. Cancer Res. 74:1945-1957.
  8. Sharma, N.L. et al. (2014). The ETS family member GABPa modulates androgen receptor signaling and mediates an aggressive phenotype in prostate cancer. Nucleic Acids Res. 10.1093/nar/gku281.
  9. Axlund, S.D. et al. (2010). HOXC8 Inhibits Androgen Receptor Signaling in Human Prostate Cancer Cells by Inhibiting SRC-3 Recruitment to Direct Androgen Target Genes. Mol. Cancer Res. 8:1643-1655 (#CBA-106-C).
  10. Alfano, R.W. et al. (2009). Matrix Metalloproteinase-Activated Anthrax Lethal Toxin Inhibits Endothelial Invasion and Neovasculature Formation during in Vitro Morphogenesis. Mol. Cancer Res. 7:452-461 (#CBA-106-C).
  11. Eckstein, N. et al. (2008). EGFR-Pathway Analysis Identifies Amphiregulin as a Key Factor for Cisplatin Resistance of Human Breast Cancer Cells. J. Biol. Chem. 283:739-750 (#CBA-106-C).